sigma-plot version 12.5 software (SPSS Inc)
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SPSS Inc
sigma-plot version 12.5 software
Sigma Plot Version 12.5 Software, supplied by SPSS Inc, used in various techniques. Bioz Stars score: 90/100, based on 1 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
https://www.bioz.com/product/sigma+plot+software+(version+12%2E5/sigma+plot+software/pm38521010-135-12-16
Average 90 stars, based on 1 article reviews
Sigma Plot Version 12.5 Software, supplied by SPSS Inc, used in various techniques. Bioz Stars score: 90/100, based on 1 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
https://www.bioz.com/product/sigma+plot+software+(version+12%2E5/sigma+plot+software/pm38521010-135-12-16
Average 90 stars, based on 1 article reviews
sigma-plot version 12.5 software - by Bioz Stars,
2026-11
90/100 stars
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Quantitative RT-PCR:Article Title: Fibrogenic Signaling Is Suppressed in Hepatic Stellate Cells through Targeting of Connective Tissue Growth Factor (CCN2) by Cellular or Exosomal MicroRNA-199a-5p Article Snippet: Analysis of Exosome-Mediated miR-199a-5p Communication in HSC Co-Cultures With the use of our previously described co-culture system, 10 , 23 exosomal delivery of miR-199a-5p was determined by assessment of dual luciferase activity in P6 mouse primary HSCs transfected with parental miR-Selection Fire-Ctx lentivector or the same vector that contained either wild-type or mutant CCN2 3′-UTR, when co-cultured for 24 hours with D1 HSCs that had been treated for 24 hours with or without 10 μmol/L of GW4869, an exosome inhibitor that blocks neutral sphingomyelinase 2, which is required for the biosynthesis of ceramide on which exosome production is dependent. Luciferase:Article Title: Fibrogenic Signaling Is Suppressed in Hepatic Stellate Cells through Targeting of Connective Tissue Growth Factor (CCN2) by Cellular or Exosomal MicroRNA-199a-5p Article Snippet: Analysis of Exosome-Mediated miR-199a-5p Communication in HSC Co-Cultures With the use of our previously described co-culture system, 10 , 23 exosomal delivery of miR-199a-5p was determined by assessment of dual luciferase activity in P6 mouse primary HSCs transfected with parental miR-Selection Fire-Ctx lentivector or the same vector that contained either wild-type or mutant CCN2 3′-UTR, when co-cultured for 24 hours with D1 HSCs that had been treated for 24 hours with or without 10 μmol/L of GW4869, an exosome inhibitor that blocks neutral sphingomyelinase 2, which is required for the biosynthesis of ceramide on which exosome production is dependent. Activity Assay:Article Title: Fibrogenic Signaling Is Suppressed in Hepatic Stellate Cells through Targeting of Connective Tissue Growth Factor (CCN2) by Cellular or Exosomal MicroRNA-199a-5p Article Snippet: Analysis of Exosome-Mediated miR-199a-5p Communication in HSC Co-Cultures With the use of our previously described co-culture system, 10 , 23 exosomal delivery of miR-199a-5p was determined by assessment of dual luciferase activity in P6 mouse primary HSCs transfected with parental miR-Selection Fire-Ctx lentivector or the same vector that contained either wild-type or mutant CCN2 3′-UTR, when co-cultured for 24 hours with D1 HSCs that had been treated for 24 hours with or without 10 μmol/L of GW4869, an exosome inhibitor that blocks neutral sphingomyelinase 2, which is required for the biosynthesis of ceramide on which exosome production is dependent. Software:Article Title: Fibrogenic Signaling Is Suppressed in Hepatic Stellate Cells through Targeting of Connective Tissue Growth Factor (CCN2) by Cellular or Exosomal MicroRNA-199a-5p Article Snippet: Analysis of Exosome-Mediated miR-199a-5p Communication in HSC Co-Cultures With the use of our previously described co-culture system, 10 , 23 exosomal delivery of miR-199a-5p was determined by assessment of dual luciferase activity in P6 mouse primary HSCs transfected with parental miR-Selection Fire-Ctx lentivector or the same vector that contained either wild-type or mutant CCN2 3′-UTR, when co-cultured for 24 hours with D1 HSCs that had been treated for 24 hours with or without 10 μmol/L of GW4869, an exosome inhibitor that blocks neutral sphingomyelinase 2, which is required for the biosynthesis of ceramide on which exosome production is dependent. |